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In line with this, a recent ex vivo study in 6 weeks bile-duct ligated (BDL) rats showed that a rise in lactate (1.7 fold) and not glutamine is an important player in the pathogenesis of brain edema in CLF [39]

They may recommend restarting at a lower dose and re-titrating to avoid a surge of GI side effects

Advancements in metabolomics, systems biology, and microbiota are also mentioned

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These defects accelerate lipid accumulation, oxidative stress, and hepatocellular injury

therefore more metabolites must be explored for direct influence on BBB physiology